School of Veterinary Medicine

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About School of Veterinary Medicine

Who works at the School of Veterinary Medicine

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Mr. Ahmed Asaid Abdelrahman Elkady
School of Veterinary Medicine - Azzaytuna University

Publications

Some of publications in School of Veterinary Medicine

تأثير الاتبان المعاملة باليوريا علي مكونات اللبن ودرجة حموظة الكرش لماعز اللبن الفرنسي ( السنن )

تهدف هذه التجربة إلي استخدام مصادر الأعلاف التقليدية من أجل توفير مصادر نيتروجينية بالأعلاف التي تقدم للحيوانات المجترة وقد أجريت هذه التجربة بمركز بحوث التقنيات الحيوية علي عشرين ( 20 ) رأس من إناث ماعز ( السنن ) لمعرفة مدى تأثير اليوريا علي مكونات اللبن و phالكرش لهذه الحيوانات حيث قسمت الحيوانات عشوائياً إلي أربع مجموعات المجموعة الأولى تبن الشعير الغير معامل ( مجموعة الشاهد ) والذي يمثل ( 40% ) من العليقة اليومية ، وللمجموعة الثانية تبن الشعير معامل بتركيز ( 2.5 % ) يوريا ، والمجموعة الثالثة تبن الشعير معامل بتركيز ( 3.5 % ) يوريا ، أما المجموعة الرابعة فقدم لها تبن شعير معامل بتركيز ( 4.5 % ) يوريا .أما ( 60% ) من العليقة اليومية فتكون من الأعلاف المركزة وأعطيت الحيوانات فترة أقلمة لمدة خمسة عشر ( 15 ) استمرت التجربة خمسة ( 45 ) يوماً، ومن خلال التحليل الإحصائي للبيانات، وجد انه لا توجد فروق معنوية عند ( 0.05>p ) بين التبن المعامل باليوريا وغير المعامل علي مكونات اللبن و PH الكرش وكان متوسط الدهن في اللبن علي التوالي كالآتي : (3.11، 3.25 ،2.94 ، 3.09 ) ومتوسط البروتين في اللبن (2.72 ، 2.76 ، 2.67 ، 2.64 ) ومتوسط PH الكرش (6.45 ، 6.56 6.69 ، 6.56 ) ومن خلال النتائج السابقة يتضح ان اضافة اليوريا الى الاعلاف الخشنة الفقيرة في البروتين الى تحسين قيمتها الغذائية.
ADEL EMHIMMID MOHAMMED SAEIDAH, , (6-2018)

(Extraction palm kernel oil from palm kernel using super-critical carbon dioxide(SC-CO2

Extraction of palm kernel oil(PKO) from dehulled ground palm kernel using supercritical carbon dioxide (SC-CO2) was studied at conditions of 313.2 and 353.2 K and at pressures from 20.7 to 48.3 MPa. The yield of PKO increased with pressure from 34.5 to 48.3 MPa at353.2K and attained a value of 49 g oil/100 g palm kernel at 48.3 MPa and 353.2 K
ADEL EMHIMMID MOHAMMED SAEIDAH, Moftah Massaud Bennama Bennama, (3-2017)

Popov Aleksandrov, A., Mirkov Ivana .,El-Muyghi, A. A. M., Glamodija Jasmin M Miljkovic Danijela Pokic J Ninkov Marina Kataranovski Dragan S & Kataranovski Milena V (2012) Differntial mechanisms of resistance to pulmonary Aspergillus fumigatus Infecti

Nitric oxide (NO) produced by inducible nitricoxide synthase (iNOS) within the central nervous system (CNS)mainly exerts adverse effects in neuroinflammation. Experimentalautoimmune encephalomyelitis (EAE), an animal model of multiplesclerosis, is a neuroinflammatory disorder. CXCL12 is a chemokine,whose antiinflammatory role in EAE has been described recently. Itsmajor producers in the CNS are astrocytes and endothelial cells ofmicro blood vessels (MBV). The aim of this work was to investigate invitro and in vivo effects of NO on CXCL12 gene expression in ratastrocytes and MBV.Materials and methods: Spinal cords (SC) were isolated from rats thatdeveloped EAE at different stages of the disease, homogenized andassessed for iNOS and CXCL12 gene expression by ‘real time’ RT-PCR.In vitro studies were performed on MBV isolated from SC of healthy,non-immunized adult rats, astrocytes derived from neonatal rats andC6 cells (rat astroglioma). After stimulation with supernatantscollected from splenocyte cultures stimulated with concanavalin Afor 48 h or with combination of cytokines (IL-1b + TNF + IFN-c + IL-17) they were exposed to NO donor, sodium-nitroprusside(SNP). Also, peritoneal cells isolated from healthy rats were co-cultivated with astrocytes. Nitrite accumulation in supernatants, asindirecte measure of NO release, was determined using Griess reactionand CXCL12 gene expression was measured by PCR. In vivo, ratsimmunized to develop EAE were treated with aminoguanidine, aninhibitor of iNOS activity, starting from day 9 post immunization,once per day until the disease peak was reached in control, untreatedgroup. At this point, CXCL12 gene expression was determined in SChomogenates and MBV.Results: We found negative correlation between gene expression ofiNOS and CXCL12 in SC of EAE rats. NO released from SNP orproduced by peritoneal cells in vitro significantly reduced CXCL12gene expression in astrocytes and MBV. In vivo inhibition of iNOS ledto upregulation of CXCL12 gene expression and reduction of EAEseverity in rats.Conclusions: These results imply that NO is important inhibitor ofCXCL12 expression within the CNS during the encephalitogenicautoimmune response. This adds to the list of harming effects of excessgeneration of NO in neuroinflammation
Amal Atia Mhfuod El- Muzghi, (9-2012)
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Dr. ADEL EMHIMMID MOHAMMED SAEIDAH

ADEL SAEIDAH is one of the staff members at the department of العام faculty of 13. He is working as a since 2017-10-22. He teaches several subjects in his major and has several puplications in the field of his interest.